Analysis and results (draft)

The results of our work suggest that the clusters of amyloid are correlated up to a certain length scale, beyond which they appear not to be correlated. In addition, the correlation length seems to grow as a function of the age of the animals, suggesting that the growth of the amyloid clusters spreads out from a nucleating center.

Current preliminary studies in human tissue samples suggest that the correlation length of the clusters are independent of the duration of the disease. This implies that by the time the disease starts, the deposition of amyloid is already in a steady state, meaning that it will not change as the age further ages.